presynaptic membrane

Results The length of postsynaptic membrane decreased obviously. The ratio of the length of postsynaptic with presynaptic membrane, and that the motor nerve terminal area with postsynaptic membrane area diminished. The AChR on the postsynaptic membrane decreased by 35.9 percent as well.

结果MG时突触后膜长度缩短，后、前膜长度比值变小，神经末端面积及其与突触后膜面积之比变小，突触后膜上的乙酰胆碱受体（AChR）减少了35.9%。

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Synaptic vesicles fuse with the presynaptic membrane upon the arrival of an action potential at the presynaptic axon terminal.

突触前轴突终末端动作电位到达时，突触小泡和突触前膜紧密相连。

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Accumulating evidence reported the function of α-SNAP in membrane fusion between vesicle membrane and presynaptic membrane, which has been regarded as the most important step in calcium-dependent exocytosis during the process of neurotransmitter release.

已有大量研究报道α-SNAP参与钙依赖的胞吐作用，该作用在神经递质释放过程中的囊泡膜与质膜的融合步骤中是至关重要的。

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The subclass analysis of IgG-AchR and presynaptic membrane receptor ( PsmR) antibodies in sera of 43 patients with myasthenia gravis was done by means of ABC-ELISA studies.

应用ABC-ELISA法分析43例重症肌无力（MG）病人血清中IgG-乙酰胆碱受体和突触前膜受体抗体亚型。

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Since t-SNARE, a key element in the formation of 20S complex, located at presynaptic membrane, we supposed that the translocation of α-SNAP might take place in the process of membrane fusion, for instance, from cytoplasm to cell membrane.

由于膜融合过程中形成20S复合体的t-SNARE位于突触前膜，所以我们推测α-SNAP在参与膜融合发生的过程中其细胞内分布有可能发生了变化，如由胞浆分布转移至胞膜。

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The postsynaptic membrane of S-, T-and G-types is apparently thicker than the presynaptic membrane and showed to be asymmetrical.

S型、T型和G型突触后膜均比前膜明显增厚，呈不对称性。

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Synapses are connections between neurons, biological signals are transferred from the presynaptic membrane by the synaptic transmission to the postsynaptic membrane.

突触是神经元之间的连接部位，生物信号从突触前膜经突触传递到突触后膜。

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Measurement of IgG and IgM anti presynaptic membrane antibody in myasthenia gravis

重症肌无力IgG型及IgM型抗突触前膜抗体检测

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Modified action of thiocyclam hydrogen oxalate on insect presynaptic membrane calcium channels

杀虫环对昆虫突触前膜钙通道的调制

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Studies on autoimmunity of myasthenia gravis I. detection of presynaptic membrane antibody ( pr mab) in myasthenia gravis

重症肌无力的自身免疫性研究Ⅰ.重症肌无力抗突触前膜抗体的检测

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A significant reduction in the number of synaptic with nuclear presynaptic membrane and postsynaptic membrane, and a low density in postsynaptic dense zone.

模型组神经元突触数量明显减少，突触前、后膜结构模糊不清，突触后致密区密度低。

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Presynaptic membrane, postsynaptic membrane and synaptic cleft were clear. Presynaptic terminals had more round clearest vesicles, and postsynaptic membrane has a high density electron. 3.

突触前膜、突触后膜和突触间隙清晰，突触前终末有较多清亮的圆形囊泡，突触后膜有电子密度高的致密物质。

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And the opening proportion of chemical-gated ion channels on postsynaptic membrane was positively related to the potential of presynaptic membrane.

突触后膜上化学门控离子通道的开放比例与突触前膜电位呈正相关；

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The presynaptic and postsynaptic membrane were damaged so seriously that the typical synaptic structure did not exist.

突触前后膜被破坏，典型的突触结构已不存在。

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Detection of Three Kinds of Antibodies to Presynaptic Membrane Binding Protein in Myasthenia Gravis

重症肌无力三种突触前膜结合蛋白抗体的检测

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Cannabinoid ( CB1) receptors are found predominantly in the brain with highest densities in hippocampus, cerebellum and striatum and mainly distributed in presynaptic membrane of never terminals and regulating the release of neurotransmitters.

研究发现CBl受体主要分布于神经系统，以脑组织中表达为主，集中分布在大脑的海马、小脑和纹状体，多定位于神经末梢突触前膜，发挥调节神经递质释放的作用。

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The readily releasable vesicles were not clustered close to the presynaptic membrane but instead were dispersed almost randomly throughout the vesicle cluster.

结果发现即刻可释放小泡并不是簇集于突触前膜而是随机分散于小泡群中。

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Excitatory amino acid transporters ( EAAT) localizes on the presynaptic membrane, synaptic vesicle and glial membrane.

兴奋性氨基酸转运体（EAAT）位于突触前膜、突触囊泡和神经胶质细胞膜上。

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Clustered microvesicles are close to and attach to the presynaptic membrane;

突触前成分内，有成群的微泡靠近并附着于突触前膜；

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To Investigate a Chronic Kindling Epilepsy Animal Model with Presynaptic Membrane Protein ( Munc-18) Antibody

神经突触前膜蛋白（Munc-18）抗体慢性点燃癫癎动物模型的探索

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Chnclusion: The features of the afferent synapses were presynaptic bodies and membrane thickening.

结论：传入突触的特征为具有突触前小体，突触膜增厚。

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Recently, transporter, that is a sort of protein located in the presynaptic membrane, vesicle membrane and glia cell membrane, has been the study hotspot of neuroscience.

近年来，一种位于突触前膜、囊泡膜及神经胶质细胞膜上的糖蛋白&神经递质转运体逐渐成为神经科学界研究的热点。

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Multiple protein components at the active zone region interact and coordinate with each other to control and regulate docking, priming and fusion of the synaptic vesicles with presynaptic membrane, and to further maintain the normal transmitter release process.

活动区内多种蛋白组分相互作用与相互协同而严格调控突触囊泡在突触前膜的入坞、触发与膜融合等过程，以保证正常递质释放过程的完成。

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